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Obeidat, A

Furthermore, H 2 can activate the MAPK/HO-1 pathway to inhibit neuronal apoptosis and alleviate ischemic brain injury in neonatal mice (Wang et al., 2020) or protect type II alveolar epithelial cells from hyperoxia-induced apoptosis by activating the PI3K/Akt signaling pathway (Wu D

(2014, February 1)

These did not show therapeutic benefit, some reasons postulated for this include crosstalk between the IGF-1R and other growth factor pathways such as the IR (Robertson et al., 2013