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Nonetheless, many studies demonstrate antioxidant properties of CoQ 10 in single cells, and benefits of CoQ 10 supplementation in humans are attributed to its ability to maintain efficient mitochondrial energy metabolism and thus prevent mitochondrial dysfunction, rather than act as a direct cellular antioxidant

A mechanistic study further revealed that E2f2 deletion in macrophages fueled proinflammatory responses in a Leu-mTORC1-dependent manner by enhancing glycolysis and impairing mitochondrial function by increasing SLC7A5 expression [105]