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To develop PROTACs for targeted degradation of RNF4, we aimed to exploit the recently described cysteine-reactive RNF4 ligand CCW16, and synthesized a set of CCW16-derived degrader molecules using established VHL- and CRBN-E3 ligands
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50 years on and still very much alive: Apoptosis: a basic biological phenomenon with wide-ranging implications in tissue kinetics
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