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But a lot of snacking comes from habit or boredom, and on a GLP-1 it can actually make it harder to eat enough at meals
[1] Its mechanism promotes insulin secretion, slows gastric emptying, and reduces appetite, leading to notable improvements in glycaemic control and body weight reduction
In the STEP clinical trials with adults who had overweight or obesity, semaglutide 2.4 mg substantially reduced CRP levels compared to placebo, regardless of starting BMI or body weight
Instead of working with your body to help your gut make more GLP-1 on its own, these medications hijack the system, giving you exponentially increased GLP-1 activity without your body actually making more of it
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