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NASH is characterized by recruitment of immune cells into the liver, mitochondrial dysfunction, endoplasmic reticulum (ER) and oxidative stress and defective autophagy, resulting in hepatocyte ballooning, apoptosis, and activation of hepatic stellate cells (HSC) 3,4
The retatrutide cost analysis becomes especially relevant here, since higher doses mean higher expenditure for incrementally smaller gains
doi: 10.1007/s00125-011-2080-1 60 LiuZStanojevicVAvadhaniSYanoTHabenerJFet al
1990), but current practice is to diagnose this change as focal fatty change rather than as a focus of hepatic alteration (Figures 12 and 13)
