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This evidence suggested that high glucose induces the activation of PSCs and promotes their proliferation, leading to increased ECM secretion
Given the ancient origin of the Ca 2+ -based signaling, which evolutionary developed earlier than the mitochondrial apoptotic toolkit whose emergence cannot be dated back to the times earlier than eukaryogenesis and acquisition of an endosymbiotic mitochondrion, it can be assumed that the Ca 2+ toolkit takes over a key role in cell death signaling in terminally differentiated mitochondria-free erythrocytes
Restriction of intramolecular motions in the aggregate state has been proposed as the main cause for the AIE phenomenon 40
doi:10.3389/fmicb.2024.1435720 40
