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On the active site thiol of gamma-glutamylcysteine synthetase: relationships to catalysis, inhibition, and regulation
Figure 1 2.3 Clinical evidence supporting the involvement of iron dysregulation in Alzheimers disease progression Recent clinical and imaging studies indicate that iron dysregulation is an active contributor to early Alzheimers disease (AD) pathogenesis rather than a mere byproduct of late neurodegeneration
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