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Beyond these real-world considerations, pharmacologic heterogeneity is biologically plausible given differences in molecular structure, receptor signaling profiles, half-life, and potency, which may influence downstream central effects (31)

The ferroptosis inhibitor liproxstatin-1 can reduce the immunosuppression of PMN-MDSCs, but protect tumor cells from ferroptosis, leading to an increased risk of metastasis (170)

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