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IGF-1 LR3 contains a 13-amino-acid N-terminal extension and a glutamic acid substitution at position 3, reducing binding affinity for IGF-binding proteins (IGFBPs) by approximately 100-fold compared to endogenous IGF-1
The 1997 Thorner review compiled the broader GHRH-and-GHRP therapeutic-research framework (PMID 9238854)
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