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MiR-7-5p is a key factor that controls radioresistance via intracellular Fe 2+ content in clinically relevant radioresistant cells
Considering that the copper-associated mitochondrial toxicity may have been delayed, we further explored the changes in the expression of skin irritation-related biomarkers at the genomic level
Studies show oxidative degradation and peptide aggregation increase beyond this window, reducing bioavailability by 1020% or more depending on storage conditions
Hydrogen tunneling in adenosylcobalamin-dependent glutamate mutase: Evidence from intrinsic kinetic isotope effects measured by intra-molecular competition
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