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Oxidative stress may then damage mitochondrial membranes and enzymes, further lowering ATP production and increasing the need for glutathione, SOD, catalase and other antioxidant defenses
soluble in aqueous buffers
It can also contribute to premature aging and skin damage
Chronic heavy drinking induces the microsomal ethanol-oxidizing system (MEOS) and CYP2E1 enzyme upregulation, increasing ethanol clearance speed while simultaneously generating reactive oxygen species that accelerate hepatocellular damage
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