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The neurological symptoms can be particularly concerning
In its role as an antioxidant, GSH effectively removes free radicals and other reactive oxygen species through the GPx activity [5], which oxidizes GSH to GSSG, and the action of NADPH-dependent glutathione reductase, which generates GSH [3]
In a number of studies, GLP-1R agonists such as liraglutide, exenatide and lixisenatide can reduce neuroinflammation in AD models, thereby improving cognitive dysfunction (140, 141, 144148)
These results show that, similar to ZnT8 LOF, ANS effectively prevents conversion of SC- cells to SC- cells in SC-islets by chemically targeting zinc accumulation
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