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Using a long-term HFD-fed mouse model of NAFLD, we demonstrated in this study that NAFLD development occurs concomitantly with dysregulated hepatic glutathione homeostasis characterized by decreased GSH and increased GSSG, and increased TNF protein levels in both the circulation and the liver

Accumulating evidence indicates that OS not only triggers inflammatory responses but also directly contributes to liver damage in ALD (Louvet and Mathurin, 2015)

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