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The progressive form, MASH, exhibits hepatocyte damage, inflammatory cell infiltration, and variable degrees of tissue fibrosis, with advanced fibrosis representing the predominant prognostic determinant for hepatic morbidity and mortality, as well as an additional risk factor for extrahepatic complications
Next, we knocked down cavolin-1 or clathrin in human caECs using a siRNA and observed no effect on cellular internalization of FAM(2836) (Supplemental Figure 6, C and D)
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