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Hence, whether TGF-1 interception of the Nrf2 antioxidant response in the diseased liver is the dominant pathogenic factor in its contribution to chronic liver disease still requires more rigorous study

It has been reported that these three compounds can enhance the contents of GSH and the activities of GSH-dependent antioxidant enzymes (GPX and GST) and reduce lipid peroxidation and cardiac damage mediated by DOX or isoproterenol (ISO) in the myocardium of rats, respectively [121, 129, 130]

Signal transduction of eNOS activation

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