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Across large RCTs and meta-analyses, these medications consistently reduce hospitalization for HF across the ejection fraction spectrum, with reductions in cardiovascular death and all-cause mortality most firmly established in HFrEF (and in chronic kidney disease and atherosclerotic disease) [6, 14, 41, 42]
From a clinical perspective, these findings underscore the value of exploring interventions that modulate and support oxidative stress pathwaysparticularly those relative to glutathione synthesis or recyclingas adjunctive strategies for managing treatment-resistant depression
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