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The main phenotype of loss-of-function mutations in arGSTs is the decreased content of anthocyanins ( 1 ) 19,20,21,22,23,24,25,26,27,28 and proanthocyanidins 27 , which can be well explained by an incomplete biosynthesis due to the absence of an essential enzyme
To evaluate the target-binding capacity of D-CuP, molecular docking simulations were conducted using AutoDock Vina against the functional domains of p53 (DNA-binding domain) and HDAC7 (catalytic deacetylase domain)
That said, the evidence suggests that 6mg remains a reasonable option for many people
Zhang Y, Yan H, Lu W, Li Y, Guo X, Xu B
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