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1998: Wessells et al
OS also reduces tetrahydrobiopterin (BH4), a critical cofactor for eNOS, causing it to produce more ROS instead of NO, thereby amplifying endothelial injury (Hernandez-Navarro et al., 2024)
It promotes actin sequestration and is studied for accelerating wound healing, reducing fibrosis, and improving function after ischemic or traumatic injuries
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