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126 Co-administration of warfarin (a substrate of CYP1A2, CYP2C9, and CYP3A4) and octreotide led to high INR levels in humans, which may be attributed to the increased warfarin exposure caused by octreotides inhibitory effect on CYP3A4

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They confirmed with the model that the Na + -dependent GSH transporter is localized at the luminal apical membrane of brain endothelial cells [37]