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Results We found that ALCAR reduces cell proliferation, induces apoptosis, hinders the production of pro inflammatory cytokines (TNF- and IFN-) and of chemokines CCL2, CXCL12 and receptor CXCR4 involved in the chemotactic axis and impairs the adhesion, migration and invasion capabilities of PCa and BPH cells in vitro

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Despite such indirect evidence suggesting the involvement of atypical Glu, Gln, and GABA levels, in vivo quantification of these metabolites in individuals with ASD has encountered substantial challenges

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Krppel-like transcription factor KLF5 is a key regulator of adipocyte differentiation

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