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Deficiency of 5-methyladenosine phosphorylase (MTAP), a key enzyme in polyamine metabolism in tumors, can accelerate the accumulation of 5-deoxy-5methylthioadenosine (MTA) in the TME, ultimately reducing the maturation of dendritic cells and the activation of T cells
Its major compound "Curcumin", acts to restore adequate glutathione levels and enhance glutathione enzyme activity
Senescent Endothelial Cell Elimination & Vascular Ageing (Frontiers in Bioengineering, 2025/2026) p53/BCL-2/Caspase-3 Pathway Activation Selectively Clears Senescent Endothelial Cells A study published in Frontiers in Bioengineering and Biotechnology investigated the mechanism by which FOXO4-DRI induces apoptosis in senescent endothelial cells
