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ORF3a-induced ROS production and cell death appears to be a highly conserved activity (312, 313), as these effects are observed in both fission yeast and human A549 and 293 T cells (287) and independent (286, 314)
Tumors can also utilize myeloid plasticity to directly manipulate dendritic cell differentiation, skewing it toward immune-suppressive macrophage-like subsets that correlate negatively with the immune response to anti-PD-L1 and anti-CTLA-4 therapies [106]
Patients showed decreased C-reactive protein levels (average reduction of 40%) and improved erythrocyte sedimentation rates
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