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Secondary carnitine deficiencies are most commonly the result of trapping in fatty acylcarnitines due to defects in mitochondrial fatty acid oxidation enzymes
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This means that the synapse mitochondria have additional functions at the nerve terminals, which may include Ca 2+ buffering directly affecting the firing probability of neurons [37, 38]
Protein expression was detected by SuperSignal West Pico Chemiluminescent substrate (Thermo, Waltham, MA, USA) and Fujifilm LAS-3000 (Fujifilm, Tokyo, Japan)
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