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The IFN-I signature characterizes an early and transient event in its progression and a link between the innate and adaptive immune response.65 Studies have shown that IFN, produced mainly by plasmacytoid dendritic cells (pDCs), stimulates the production of chemokines, such as CXCL9 and CXCL10, by keratinocytes, leading to the recruitment of T-cells expressing their receptor: CXCR3.65,72 Furthermore, HSP70 potentially contributes to skin inflammation by activating pDCs and increasing IFN secretion.72 Cytotoxic CD8+ T-cells are necessary and sufficient for the destruction of melanocytes, acting as an effector arm of autoimmunity.78 The lesions are caused by effector CD8+ T-lymphocytes in the initial or active phase of the disease and by recirculating and resident memory CD8 + T (TEM) lymphocytes in the stable phase

Such subtle but widespread alterations of conduction velocity, which would perturb network synchronization in the brain, could be the substrate of frequent and disabling non-focal MS symptoms such as fatigue, mood disorders and cognitive decline (Kalb et al., 2019

Compounded drugs are not directly approved by the FDA in the same way as commercial drugs

Magnesium and B vitamins improve glutamine metabolism and gut cell regeneration