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Growth hormone receptors are present on many cell types involved in wound healing, including fibroblasts that produce collagen and keratinocytes that regenerate the skin surface
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Several peptides being studied have mechanisms that directly address this core problem: they promote new blood vessel formation (angiogenesis), reduce the inflammatory cascade that accelerates disc breakdown, and stimulate the cellular repair processes needed to rebuild damaged disc structures
How it works KPV blocks the inflammation switch from reaching the cell nucleus, lowering production of inflammatory molecules (NF-B signaling)
