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Regarding GLP-2, Gu and colleagues evaluated a modified GLP-2 analog in mice, demonstrating that it reduced disease severity by restoring epithelial junctions and barrier integrity (87), complementing their currently approved use of GLP-2 analogs in short bowel syndrome (88, 89)
These changes compromise granulosa cell support, impair meiotic progression, and accelerate ovarian aging, contributing to female infertility disorders such as polycystic ovary syndrome
