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This upregulation reduces the sensitivity of nasopharyngeal carcinoma (NPC) cells to ferroptosis, thereby fostering chemoresistance and tumor progression ( Figure 6 Overall, as a carcinogenic virus, EBV shares similar mechanisms with HBV, both of which facilitate tumor progression and induce chemotherapy resistance by suppressing ferroptosis sensitivity in tumor cells
Exogenous substances like N-acetylcysteine can raise the intracellular amounts of GSH [110]
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Fas-stimulated generation of reactive oxygen species or exogenous oxidative stress sensitize cells to Fas-mediated apoptosis
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