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Gene therapy for articular cartilage repair
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However, some meiotic cells were still present in the gld-2 gld-1 prom-1 triple mutant, suggesting that there may be an additional pathway(s) that functions redundantly with the GLD-1, GLD-2 and SCF PROM1 pathways to promote meiotic entry and/or inhibit proliferation (Mohammad et al., 2018)
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