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In this review we sum up the latest information regarding the aetiology and therapy of IIH

Our previous data also suggests that maternal SE impairs brain mitochondrial levels of oxidative phosphorylation (OXPHOS) complexes in male offspring with reduced endogenous antioxidant capacity from birth (Chan et al., Furthermore, as SE offspring have reduced brain antioxidants and increased cellular oxidative damage (Chan et al., 2015) and can provide neuroprotection by elevating brain antioxidant capacity in aging rats (Juliet et al., 2015)

Post translational modulation of -Amyloid precursor protein trafficking to the cell surface alters neuronal iron homeostasis

364 Emodin can relieve collagen-induced joint inflammation in rats by inhibiting ferroptosis and degrading MMP