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These drugs work by mimicking the action of naturally occurring GLP-1, an incretin hormone that stimulates insulin secretion in response to food intake, suppresses glucagon release, and slows gastric emptying

Although they are inhibited by GPX4 inhibitors such as RSL3 in vitro , treatment of cells with more specific TrxR inhibitors, e.g., TRi, does not induce ferroptosis [102] indicating that TrxR is not directly involved in ferroptosis

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