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65, 66 Third, we found in both the pancreatitis models a marked overexpression of GLP-1R, which generally colocalized with activated PSCs or with areas of marked increased in PSC activity in the CP model with enhanced fibrosis
The point is not to be anti-medication
As our understanding on OSA pathophysiology expands, we can anticipate a more diverse range of effective treatments, enabling personalized therapeutic approaches tailored to individual patient symptoms and long-term outcomes
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