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While pharmacologic inhibition may not induce enough inhibition, G6PD knockdown may go too far, and neither recapitulates the protein instability that is characteristic of human G6PD-d

The study indicates this may not be because the patients aren't producing enough glutathione, it may be because glutathione is being used at a higher rate in patients with type II diabetes

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In tumors, persistent ROS elevation increases GSH synthesis and shifts the redox balance, creating a biochemical environment that selectively activates disulfide/diselenide linkers and metal-based GSH-depleting nanoplatforms