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Regulation of gamma-glutamyl-cysteine synthetase by nonallosteric feedback inhibition by glutathione
Thus, we can conclude that baicalein treatment confers neuroprotection against post-traumatic seizures, oxidative damage, neuroinflammation and neuronal apoptosis in animal models of TBI
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GSH intensity in the GCL, but not inner nuclear layer (INL), of EAAC1 KO mice was significantly lower than that of WT, and NAC treatment suppressed the GSH reduction in the GCL (Fig
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