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Targeting the renin-angiotensin-aldosterone system in fibrosis
Furthermore, since 2-HG is structurally similar to -KG, it competitively inhibits some -KG-dependent dioxygenases, including the ten eleven translocation enzymes (TETs) and the Jumonji family of histone lysine demethylases, giving rise to DNA and histone hypermethylation [38, 39]
Mechanistic rationale to target PTEN-deficient tumor cells with inhibitors of the DNA damage response kinase ATM
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