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4e) and higher accumulation of mitochondrial mass, probably due to prevention of ROS-induced organellar damage (Fig
Future studies should validate these targets in preclinical models and explore their translational potential to develop interventions improving the outcomes of patients with SAE
Secondly, restoring optimal glutathione levels directly supports the liver's Phase II detoxification pathways
GPX2 and GPX3 exert antioxidant effects in intestinal epithelial cells and plasma, respectively [65]
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