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Thus, it has been suggested that the hyperglucagonaemia observed with type 2 diabetes may be owing to concurrent liver disease, specifically hepatic steatosis, causing impairment of the liveralpha cell axis (owing to desensitised hepatic GCGR signalling), resulting in hyperaminoacidaemia and, thereby, increased plasma glucagon both in the fasting and postprandial state [99]
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PMID 11239739
doi:10.1152/jappl.1976.41.4.523
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