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Example: models of pelvic inflammatory disease show reduced lesion size and inflammatory infiltrate after T4 treatment

Sustained oxidative stress further induces increased expression of pro-apoptotic proteins such as BAX and reduced expression of BCL-2, thereby exacerbating programmed cell death in both epididymal and testicular germ cells (64)

ACE-inhibitory peptides potentially block the active site of ACE and diminish the conversion of angiotensin I to angiotensin II, thus alleviating blood pressure [117]

This is consistent with the next comparison among subpopulations pointing to altered pathway activity in MAC.2 ( Figure 5D )