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This means that it binds to the same receptors as does endogenous ghrelin (the GHSR-1a receptors) and that it stimulates the cells in the pituitary gland to release more GH without raising ACTH or cortisol levels [3]
du Cheyron D, Daubin C, Poggioli J et al (2003) Urinary measurement of Na+/H+ exchanger isoform 3 (NHE3) protein as new marker of tubule injury in critically ill patients with ARF
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