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By preventing iron-mediated oxidative damage, GHK-Cu protects tissues during the vulnerable post-injury period when normal iron homeostasis is disrupted
Chavakis, E
This suggests that there might be different lipid metabolic pathways in the two subtypes of HF, and inhibiting myocardial energy deficiency, by restoring ATP-dependent LV relaxation and contraction, might cause beneficial effects in HFpEF [7]
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