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Exosomes derived from synovial mesenchymal stem cells (MSCs) of patients with RA, which exhibit highlevel expression of miR146a, can specifically target and IL17mediated proinflammatory signals and TLR pathways, thereby reducing the invasiveness of synovial fibroblasts [454]
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This molecule also reduces the depletion of central glutathione and the activity of GPx and superoxide dismutase (SOD) caused by acrylonitrile while decreasing the levels of nitric oxide, MDA, interleukin-6, and TNF- that are raised by neurotoxins
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