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TB4 and, by extension, TB-500 may work on a cellular level to reduce inflammation by regulating autophagya process of cellular waste disposal [28]
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81 Additionally, DLST-mediated succinylation of PDHA1 at K83 enhances PDH activity and -KG accumulation, which activates OXGR1MAPK/ERK signaling in TAMs, suppressing MHC-II expression and T-cell priming
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