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Oxidative stress-induced mitochondrial DNA damage in human retinal pigment epithelial cells: a possible mechanism for RPE aging and age-related macular degeneration
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Specifically, genetic knockout of Gasdermin D (GSDMD/) or treatment with the GSDMD inhibitor Disulfiram strictly prevents the packaging of GSDMD-N-expressing mitochondria into microvesicles, subsequently abolishing microvesicle-induced NETosis and protecting against sepsis-induced acute injury
Those who were moved to a placebo regained some weight even with continued lifestyle changes
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