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The reason is mechanistic: Ipamorelin acts as a selective agonist of the ghrelin receptor (GHS-R) and stimulates GH release through a different regulatory pathway than GHRH
Patients who reduce refined carbohydrates and dietary saturated fat while on GLP-1 treatment tend to see stronger lipid improvements than those who rely on the medication alone
For example, pyruvate kinase muscle isoform 2 (PKM2) inactivation can both attenuate the glucose flux to PPP and enhance folate metabolism to mediate NADPH generation
This is the most common thing patients say in their first month on GLP-1 therapy
