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Interestingly, we also found that the hypoxia-induced decline in PP2A activity was reversed when -syn or p--syn was interfered with, suggesting that hypoxia-induced PP2A inhibition and -syn pathology are mutually reinforcing vicious cycles

doi:10.7554/eLife.02200 Kenzelmann Broz D, Spano Mello S, Bieging KT et al (2013) Global genomic profiling reveals an extensive p53-regulated autophagy program contributing to key p53 responses

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Similar content being viewed by others Subjects Introduction Many cells respond to injury by secreting proinflammatory cytokines and chemokines, such as tumor necrosis factor-alpha (TNF-), interleukin (IL)-1, IL-6, and CC motif chemokine ligand 2 (Ccl2) 1,2,3,4,5,6