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This type of compound undergoes GST-catalyzed breakdown to release locally cytotoxic metabolites and thereby attenuate off target adverse side effects

Similarly, GHRP-2 appears to engage ghrelin receptors, with studies suggesting downstream signaling through phospholipase C, inositol trisphosphate (IP3), and diacylglycerol (DAG)

Nutr Neurosci 22(3):145155

Conventional gastroprotective drugs commonly prescribed to minimize NSAID toxicity or heal NSAID-induced ulcers include H2-receptor antagonists (cimetidine, ranitidine, famotidine), proton pump inhibitors (omeprazole, lansoprazole, esomeprazole), and misoprostol (a synthetic prostaglandin analog) (Vonkeman 2010)