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Botulinum toxin a attenuates osteoarthritis development via inhibiting chondrocyte ferroptosis through slc7al1/gpx4 axis
Feist Cardiovascular Research Fellowship to RP and the Endowed Chair in Diabetes to SKJ from the Center for Cardiovascular Diseases and Sciences (CCDS), LSUHSC, Shreveport, as well as grants to SKJ from the National Institutes of Health/National Center for Complementary and Integrative Health (RO1 AT007442, 2013-16)
Consistent with our hypothesis of glycolytic flux as a requirement for cellular protrusion and migration, Kondo et al
Athletes using BPC-157 for chronic tendinopathy or post-surgical recovery represent the largest patient group at BHRC
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