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From KPV, a tripeptide that directly suppresses the NF-kB pathway central to psoriatic inflammation, to GHK-Cu, a copper peptide that modulates cytokine production while simultaneously supporting tissue repair, researchers are identifying mechanisms that address psoriasis at its immunological roots rather than merely suppressing surface symptoms

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Less necrosis developed in treated wounds, suggesting the peptide helped preserve marginally viable tissue that might otherwise have died