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However, overactivation of iron-related autophagy leads to iron overload in cells and increases the sensitivity of cells to ferroptosis, resulting in depletion of glutathione and a reduction in GPX4, which is the most important ROS scavenging compound in the body, ultimately leading to ferroptosis
In contrast, ferroptosis inhibition via iron chelation or lipid radical scavenges blocked this Hmox1 activation-induced cardiac damage, supporting a ferroptosis targeting therapeutic strategy to alleviate cardiomyopathy in SCD 46
Levothyroxine: Separate administration by at least four hours to avoid reduced absorption
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