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Published 2025 Aug 6

This finding reveals that targeting the SMAD2/STAT3 palmitoylation cycle can inhibit Th17 differentiation, providing a new therapeutic strategy for multiple sclerosis (152)

Notably, the hnRNP-E1 level was higher in iPSCs, likely reflecting their characteristically larger nuclear-to-cytoplasmic ratio associated with pluripotency, where nuclear proteome represents larger fraction of the total cellular proteome