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Impairment of lipophagy by PNPLA1 mutations causes lipid droplet accumulation in primary fibroblasts of Autosomal Recessive Congenital Ichthyosis patients
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The results: 35% reduction in body mass 30% decrease in adipocyte size 40% decrease in adipocyte volume No change in food intake (meaning the weight loss was purely metabolic) Cholesterol levels normalized to those of lean mice This is fundamentally different from appetite suppressants like GLP-1 agonists
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